Related Subjects:
|Classical Ventricular Tachycardia
|Idiopathic Ventricular Tachycardia
|Right Ventricular Outflow Tract Tachycardia
|Idiopathic Fascicular Left Ventricular Tachycardia
|Left Ventricular Outflow Tract Tachycardia
|Ventricular Fibrillation
|Resuscitation - Adult Tachycardia Algorithm
|Resuscitation - Advanced Life Support
|Automatic Implantable Cardioverter Defibrillator (AICD)
⚡ Idiopathic ventricular tachycardia (IVT) in patients with an anatomically normal heart is a distinct entity.
Its management and prognosis differ from VT associated with structural heart disease.
👉 RVOT VT is the commonest form of idiopathic VT, and should always be distinguished from ARVC/ARVD.
📖 About
- A form of Idiopathic Ventricular Tachycardia (IVT).
- ~80% of IVT cases arise from the Right Ventricular Outflow Tract (RVOT). 🫀
- ECG: Typically shows LBBB morphology because the arrhythmia originates in the RV. 📉
🧬 Aetiology & Mechanism
- Occurs in the absence of structural heart disease ✅.
- Usually due to a re-entrant or triggered activity mechanism involving the RVOT.
- Classically non-sustained, repetitive, and monomorphic VT.
- Triggered by exercise, stress, or catecholamine surges (adrenergic-dependent). 🏃♀️💉
✅ Diagnostic Criteria
- No structural heart disease (confirmed by Echo/CMR).
- No metabolic or electrolyte abnormalities (exclude hypokalaemia, hypomagnesaemia, thyroid). 🔬
- No inherited channelopathy (e.g., Long QT, Brugada, CPVT).
📊 RVOT VT ECG Example
👩⚕️ Clinical Features
- Commoner in females, typically aged 30–50 years 👩.
- Paroxysmal, exercise-induced sustained VT episodes. 🏋️♀️
- Symptoms: Palpitations 💓, presyncope/syncope 😵, occasional chest tightness.
- Often well tolerated but recurrent → lifestyle impairment.
🧪 Investigations
- 🧾 Bloods, Echo, CXR: Normal.
- 📉 ECG during VT: LBBB morphology + inferior axis (due to RVOT origin).
→ rS in V1 and tall R in V6 are typical.
👉 Key exam pearl: ARVD/ARVC differs - resting ECG often shows T-wave inversion in V1–V3 ± epsilon wave.
- 🏃♀️ Exercise stress test: May provoke VT (adrenergic trigger).
- 🧲 MRI: Can show subtle RV wall abnormalities in up to 70% (but be cautious - overlaps with ARVD).
🚨 Classical VT is still the most common cause of wide-complex tachycardia.
There is no completely reliable way to distinguish classical VT from idiopathic VT or SVT with aberrancy by surface ECG alone.
👉 Always treat as VT until proven otherwise.
💊 Management
- 🔑 First rule: If diagnosis not certain ➝ treat as classical VT per ALS Adult Tachycardia algorithm.
- ABC + early Echo to confirm LV function.
- 🏥 If stable RVOT VT confirmed ➝ may terminate with Adenosine 6–24 mg (diagnostic & therapeutic). ❗ Not effective in ARVD.
- Verapamil IV (10 mg over 3–5 min) can be effective, but only if Echo confirms normal LV function.
⚡ Be prepared for DC cardioversion if patient deteriorates.
- Beta-blockers (especially non-selective) may suppress adrenergic-triggered VT.
- 💡 Radiofrequency ablation (RF ablation) offers curative treatment with >85–90% success rates for recurrent symptomatic RVOT VT.
📚 References